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In addition to inducing insulin release, GLP-1R agonism can improve function and prevent exhaustion of pancreatic β cells on a transcriptional level by stimulating multiple pro-proliferative and anti-apoptotic pathways, while optimising intracellular metabolism (Figure 1)
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Quantity:10 Vials
To view a copy of this licence, visit About this article Cite this article Godschall, E.N., Gungul, T.B., Sajonia, I.R
